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Benjamin Rost

(PI; Charité, Berlin)

Project(s) involved: Hegemann/Rost

 

Curriculum vitae Benjamin Rost

German Center for Neurodegenerative Diseases, (DZNE)

Charitéplatz 1, CCO

D-10117 Berlin

Tel: +49-30-450 639080

Fax: +49-30-450 539943

E-Mail: benjamin.rost@dzne.de

Nationality: German              

Date of Birth: 24.3.1980

 

2014-15      Four month parental leave

2014           Grass fellow at the Marine Biology Laboratory, Woods Hole, Ma, USA

since 2013  Postdoc at the German Center for Neurodegenerative Diseases, (DZNE), laboratory of Prof. Dietmar Schmitz, Charité Berlin

2012-13      Postdoc in the laboratory of Prof. Christian Rosenmund, Neurocure / CCO, Charité Berlin

2006-11      PhD Thesis in the Neuroscience Research Centre, laboratory of Prof. Dietmar Schmitz, Charité Berlin

2005           Diploma / MSc. in biomedical science. Dept. of Pharmacology, Prof. Gudermann

2000-05      Studies in Humanbiologie (biomedical science), Philipps-University Marburg, Germany

 

Awards

2014           Grass fellowship for research stay at the Marine Biology Laboratory, Woods Hole, MA, USA

2009           1st Price for Student’s Talks, Berlin Brain Days 2009

2008           2nd Price for Poster Presentations, Berlin Brain Days 2008

2008-10      associated Fellow, Graduate Program 1123 (GRK1123), German Research Foundation (Deutsche Forschungsgemeinschaft, DFG)

2007           Fellow, PhD Program Medical Neuroscience, Charité Berlin

2001-10      Scholarship and Fellow, e-fellows.net, Germany

2000-05      Scholarship and Fellow, Studienstiftung des deutschen Volkes (German National Merit Foundation)

 

Publications (selection)

Rost BR*, Schneider F*, Grauel MK, Wozny C, Bentz C, Blessing A, Rosenmund T, Jentsch TJ, Schmitz D, Hegemann P, and Rosenmund C. 2015. Optogenetic acidification of synaptic vesicles and lysosomes. Nature Neuroscience Advance Online Publication 2015/11/09.

Koo SJ, Kochlamazashvili G, Rost BR, Puchkov D, Gimber N, Lehmann M, Tadeus G, Schmoranzer J, Rosenmund C, Haucke V, and Maritzen T. 2015. Vesicular Synaptobrevin/VAMP2 Levels Guarded by AP180 Control Efficient Neurotransmission. Neuron 88(2):330-44.

Watanabe S, Trimbuch T, Camacho-Perez M, Rost BR, Brokowski B, Sohl-Kielczynski B, Felies A, Davis MW, Rosenmund C, and Jorgensen EM. 2014. Clathrin regenerates synaptic vesicles from endosomes. Nature 515(7526):228-33.

Watanabe S, Rost BR, Camacho-Perez M, Davis MW, Sohl-Kielczynski B, Rosenmund C, and Jorgensen EM. 2013. Ultrafast endocytosis at mouse hippocampal synapses. Nature 504(7479):242-7.

Kaden D, Harmeier A, Weise C, Munter LM, Althoff V, Rost BR, Hildebrand PW, Schmitz D, Schaefer M, Lurz R, Skodda S, Yamamoto R, Arlt S, Finckh U, and Multhaup G. 2012. Novel APP/Abeta mutation K16N produces highly toxic heteromeric Abeta oligomers. EMBO Mol Med 4(7):647-59.

Rost BR, Nicholson P, Ahnert-Hilger G, Rummel A, Rosenmund C, Breustedt J, and Schmitz D. 2011. Activation of metabotropic GABA receptors increases the energy barrier for vesicle fusion. J Cell Sci 124(Pt 18):3066-73.

Rost BR, Breustedt J, Schoenherr A, Grosse G, Ahnert-Hilger G, and Schmitz D. 2010. Autaptic cultures of single hippocampal granule cells of mice and rats. Eur J Neurosci 32(6):939-47.

Harmeier A, Wozny C, Rost BR, Munter LM, Hua H, Georgiev O, Beyermann M, Hildebrand PW, Weise C, Schaffner W, Schmitz D, and Multhaup G. 2009. Role of amyloid-beta glycine 33 in oligomerization, toxicity, and neuronal plasticity. J Neurosci 29(23):7582-90.

Sel S*, Rost BR*, Yildirim AO, Sel B, Kalwa H, Fehrenbach H, Renz H, Gudermann T, and Dietrich A. 2008. Loss of classical transient receptor potential 6 channel reduces allergic airway response. Clin Exp Allergy 38(9):1548-58.

Motazacker MM, Rost BR, Hucho T, Garshasbi M, Kahrizi K, Ullmann R, Abedini SS, Nieh SE, Amini SH, Goswami C, Tzschach A, Jensen LR, Schmitz D, Ropers HH, Najmabadi H, and Kuss AW. 2007. A defect in the ionotropic glutamate receptor 6 gene (GRIK2) is associated with autosomal recessive mental retardation. Am J Hum Genet 81(4):792-8.

 

* equal contribution